In recent years, Autistic Spectrum Disorder (ASD) has shown a dramatic increase in prevalence. A review of prevalence survey research for ASD (identified by DSM-IV criteria for Autism, Asperger’s Syndrome, and Pervasive Developmental Disorder-Not Otherwise Specified) across the United States and the United Kingdom reported rates of ASD substantially increased from prior surveys indicating 5 to 10 per 10,000 children to as high as 50 to 80 per 10,000 (equivalent to a range of 1 in 200 to 1 in 125 children with ASD) (Blaxill, 2004). Another review of research on the epidemiology of Autism (Medical Research Council, 2001) indicated that approximately 60 per 10,000 children
(equivalent to a range of 1 in 166 children) are diagnosed with Autistic Spectrum Disorder.
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Previous EEG/QEEG Findings
• Cantor et al. (1986) – Maturational delay, including excess slow wave activity, greater coherence and less assymetry.
• Ogawa et al. (1982) – Signs of diminished hemispheric lateralization, excessive bilateral frontal alpha activity.
• Dawson et al. (1982) – EEG signs of reversed lateralization in 30%.
• Hashimoto et al. (2001) – Spike discharges frequent in frontal regions.
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Research on autistic spectrum disorder (ASD) has shown related symptoms to be the result of brain dysfunction in multiple brain regions. Functional neuroimaging and electroencephalography research have shown this to be related to abnormal neural connectivity problems. The brains of individuals with ASD show both areas of excessively high connectivity and areas with deficient connectivity. This article reviews emerging evidence that neurofeedback guided by connectivity data can remediate these connectivity anomalies leading to symptom reduction and functional improvement. This evidence raises the hopes for a behavioral, psychophysiological intervention moderating the severity of ASD. Both empirical data and a case example are presented to exemplify this approach.
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This article looks at the experience of Frankie, an autistic 8 and 1/2 year old boy. He was diagnosed mildly autistic by several specialists. One specialist claimed he was brain damaged and "autistic-like " and that there was no hope for improvement. At Frankie's mother's request, neurotherapy diagnosis and treatment was begun. After 31 sessions, Frankie showed Positive changes in all the diagnostic dimensions defining autism in DSM-111-P, This has profound implications for treatment in a field with few low-risk alternatives.
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ScienceDaily (Feb. 28, 2008) — Research on autistic spectrum disorder (ASD) shows that neurofeedback (EEG biofeedback) can remediate anomalies in brain activation, leading to symptom reduction and functional improvement. This evidence raises the hopes for a behavioral, psychophysiological intervention moderating the severity of ASD.
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